Climacteric neurosis and ways of its non-hormonal treatment

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When harmoniously functioning of humoral and neural mechanisms of adaptation menopause is painless. When adaptation is developing decompensated menopause occurring as a disease for a long time, or menopause with unstable compensation, host painful for only when the development of additional adverse conditions (exacerbation of concomitant diseases, fatigue, mental shock, and so on).
Modern ideas about the nature of menopause should determine the choice of therapeutic agents used for treatment of his pathological manifestations. Pathogenetic conditionality therapeutics should proceed from the fact increasing the activity and loss of rhythmic functioning of hypothalamic centers, increasing the activity of the reticular formation, reducing the mobility of nervous processes. To achieve the desired effect is possible through various means, including both medical (including hormonal)and physiotherapeutic methods of influence. That is why currently the most commonly used complex therapy, which consists of the combined use of estrogen, androgen, and some sedatives.
Since the objective of this Chapter is to provide an overview of non-hormonal methods of treatment of pathological menopause, we state mainly these therapies, without belittling the significance and hormonal methods, all the more that the use of androgens and estrogens, as will be shown later, also affects the centers of the hypothalamus.
Detected reduction lability pathological menopause can be considered that the success of therapy depends on what kind in respect of influence on the level lability particular tool, one or the other method. If they raise lability - they should lead to recovery, if lower lability - they lead to deterioration.
Are there any data allowing to identify, in advance, how does something on the level lability?
The Works Of N. That is, Vvedensky, A. A. Ukhtomsky, L. L. Vasiliev was shown that the majority of drugs, organic poisons, cooling, ultraviolet irradiation, ezerin, potassium cations, the cathode DC lead to lower lability. Divalent ions of calcium, barium, acetylcholine, novocaine, moderate warming, bromine soda, caffeine, vitamin B1, infrared rays, the anode DC increase lability. The action of the agents listed groups clearly revealed only at application of moderate doses (L. L. Vasil'ev, E. T. Blagodatov, 1961).
Subsequent research has also revealed that estrogens, androgens, vitamins E, C, chlorpromazine, reserpine, being applied in therapeutic doses, also increase lability.
Thus, knowing the influence of those or other means on the level lability, it is possible to foresee their action in climacteric neurosis.
The most detailed study in this regard has been subjected method anemicescoy galvanization brain.
The history of the question of the effect of direct current at the micro-organism has more than 170 years: in 1802 Ritter first tested the influence of a direct current, holding in hands the electrodes from the battery galvanic elements. In subsequent years, due to research of the Russian physiologists have been clarified important details of the clinical effects of anode DC, which was delivered to the brain (N. That is, Vvedensky, the Baltic Fleet Verigo, V. S. Rusyns, D. A. Lapitsky, and others).