Dermatitis is a skin diseases (mainly inflammatory)arising under the influence of exposure to a variety of external factors (physical, chemical, mechanical and other). The presence of exogenous stimulus is the main condition for the emergence of dermatitis, unlike taxidermy (see).
There are acute and chronic forms of dermatitis. A characteristic feature of acute dermatitis is the occurrence of him in the application of the stimulus, following the termination of which starts the development of the inflammatory process. Dermatitis is accompanied by a burning sensation, pain, fever, itching. Leading clinical symptoms are marked swelling and redness, sharply distinguished from healthy skin, no tendency to spread. Against this background may appear bubbles or bubbles; skrivas, they form erosive areas, the surface of which is shrinking in peel. Under the action of high or low temperatures, strong acids may develop necrosis followed by the formation of a scab, ulcers, scar.
Chronic dermatitis caused by prolonged exposure to relatively weak stimuli (friction tyre, corset, scratches due to itching and slight redness, pigmentation, minor infiltration of the skin thickening her and desquamation.
Dermatitis are artifactual (simple) and allergic.
Artifactual dermatitis arise immediately from exposure to obligate stimuli, i.e. such that necessarily cause dermatitis (for example, electric current, strong acids or alkalis and other).
Allergic dermatitis occur only in people with hypersensitivity (sensitization) to various stimuli (drug or the chemicals, sun, and others). Such stimuli called elective (optional)as allergic dermatitis develops only when they are repeated skin exposure, due to the absence of organism (see Allergy). Clinically for allergic dermatitis characterized ackzemopodobne picture and long incubation period (from 5-6 days to several weeks). Can occur secondary allergic rash.
Actinic dermatitis arise from the action of ultraviolet rays (solar or artificial). In the open areas of skin after 3 to 6 hours. after the action of sunlight appears edema erythema, accompanied by a stinging sensation or itching and peaking within 12-24 hours. In more severe cases on erythematous background bubbles and blisters, after opening the form of erosion, leaving after healing pigmentation. You may experience necrotizing ulcerative areas, healing scar; often there are General disorders: malaise, fever, etc. Repeated and long-term (years) exposure to the sun even in people with no sensitivity to ultraviolet rays cause skin changes in open areas (face, neck, forearms and rear brushes): persistent pigmentation, dryness, loss of elasticity, wrinkles.
Treatment: in cases of acute inflammation is applied boric, resorcinol or other gadgets (see) within 2-3 days, then corticosteroid ointments (sinalar, oksikort and others); in the absence soak and edema - cream Unna (lanolin, petrolatum on 20,0 g, distilled water 10,0 g), corticosteroid ointments. Prevention: people with increased skin sensitivity to sunlight should avoid exposure to sunlight, wear a wide-brimmed hat, use protective ointments with quinine, the salol, para-aminobenzoic acid, creams "Luch", "Spring", etc. When using UV rays for medical purposes pre-determine the sensitivity of the skin by biodosimetry.
Dermatitis from electric current characterized by necrosis of tissues in the scope of current, progressing for several days after the electric shock, lack of inflammatory reaction, painlessness, slow and sluggish over. Hair remains unaffected. The principle of treatment is to protect affected skin from any external influences, therefore, apply frame dressing. Surgical resection of necrotic masses, as well as bandages and ointments contraindicated.
Prevention: technical aspects of protection when working with electrical shock.

  • Dermatitis medication
  • Dermatitis traumatic or mechanical
  • Dermatitis from chemicals
  • Dermatitis from plants