Radiation damage to the skin

Radiation damage to the skin, often called radiation burns, can have various clinical manifestations (Fig. 5-10).

Fig. 5-10. Radiation damage to the skin (the development of radiation burns). Fig. 5. Erythema. Fig. 6 - 8. The development of bubbles. Wet RadioEthernet. Fig. 9. Erosion. Fig. 10. The scar; the visible dyschromia, telangiectasia and hem hyperpigmentation.

Erythema - temporary redness at the site of exposure; develops on 13-14-th day after single and through 2-6 weeks after the fractional irradiation.
Persistent hair removal develops at one time or fractional irradiation of the scalp. Dry epidermic develops within 7-10 days after a single or 2-3 weeks after the fractional irradiation. Clinically manifested by erythema, swelling of the skin, followed plate peeling. Restoration of irradiated skin incomplete. The skin remains dormant, dry, apriranno. Later appear telangiectasias, uneven pigmentation.
Wet RadioEthernet is accompanied by redness and swelling of the skin, the appearance of blisters filled with clear yellowish liquid, which quickly opened, thus exposing the basal layer of the epidermis. After 1-2 days begins epithelization.
Wet epidermis ends stand atrophy hair follicles, sebaceous and sweat glands, significant thinning of the skin, loss of elasticity, depigmentation (dyschromia), the emergence of telangiectasias. Later can be identified hyperkeratosis (excessive keratinization) and sclerosis subject subcutaneous fat. After irradiation of the hard x-ray or gamma radiation after 6-9 months. and later revealed slowly progressive atrophy of muscle tissue and bone osteoporosis. The most dramatic degree of muscle wasting and stunting bones observed in children.
In the treatment of malignant tumors wet RadioEthernet valid only on small fields exposure.
Radiation ulcer may develop acute in the coming days and weeks after intensive one-time exposure, subacute through 6-10 weeks, and several years after exposure. Acute for intensive reddening of the skin soon after exposure, accompanied by acute edema, severe pain, a violation of the General conditions. On edema, congestive hyperemia skin appear large bubbles often with hemorrhagic turbid contents. Upon rejection of the epidermis exposed nekrotizirovanne surface nasnimali bloom in the center of which is a plague. For a long time is the rejection of necrotic tissue, education sluggish and weak to the granulation and epithelization of ulcers. Often healing does not occur. Subacute developing radiation ulcer is often the outcome of long-flowing wet epidermica. Surrounding the ulcer tissues within the irradiated field in the next few months, develops pronounced ray atrophy.
Late radiation ulcer usually develops on the background of sharply atrophied tissues at the site of exposure. Ulceration occurs by type of acute radiation necrosis of tissues in the entire field of radiation, exciting not only the skin but also the underlying tissues, subcutaneous tissue, muscles, bones. In some cases, on atrophied skin surface appears excoriations (abrasion), which gradually deepened and enlarged, turning into a deep ulcer.
Radiation skin atrophy and radiation ulcers often end with the development of radiation cancer.
The result of radiation exposure of the skin and subcutaneous fatty tissue is often indurativnyy tissue swelling.
Indurativnyy edema develops as a result of defeat not only the blood, and lymph vessels, which leads to the violation of lymphatic circulation, swelling and sclerosis of the skin and subcutaneous tissue. Skin and subcutaneous tissue irradiated field gradually become dense, rise above the level of the normal skin, with pressure remains hole. Leather hyperpigmentation covered with telangiectasia or becomes reddish-purple hue becomes painful. Under the influence of injury or for no apparent reason in the area indurativnyy edema may occur skin necrosis, leading to the formation of deep beam ulcers.

  • The treatment of radiation damage to the skin