Peritonitis - inflammation of the peritoneum. May be specific (tuberculosis) and nonspecific etiology. Nonspecific peritonitis develops as a result of getting into the abdominal cavity of microorganisms (Streptococcus, Staphylococcus aureus, Escherichia coli, Streptococcus pneumoniae, and others) or from the effects on the peritoneum toxic substances. In the vast majority peritonitis develops secondarily as a complication of purulent diseases of perforation or damage to the organs of the abdominal cavity. Much less peritonitis caused by infection transmission by blood or lymph from some distant inflammation (tonsillitis, pneumonia and other). In those cases, when the source of peritonitis cannot be established, say cryptogenic, or asochakova, peritonitis. The impact on the peritoneum irritating factors (chemical, thermal and mechanical) can lead to the development of aseptic peritonitis.
Depending on the prevalence of inflammatory process distinguish peritonitis local and General (bottled), limited and unlimited, and on the nature of the flow - acute and chronic.
Reaction peritoneal on the introduction of microorganisms is the allocation of an inflammatory exudate, serous or purulent. The presence of exudate of fibrin leads to rapid sticking intestinal loops with a seal, with parietal peritoneum and among themselves.

Peritonitis (peritonitis; from the Greek. зуritonaion - peritoneum) - inflammation of the peritoneum in the result of penetration into the abdominal cavity of microorganisms or from the effects on the peritoneum toxic substances.
The peritoneum (see) is actively functioning organ. It singles out and sucks tissue fluid.
The peritoneum can suck suspended solids, bacteria, colloids, crystalloids. It handles most protective function: cell mesotheli are active phagocytes, peritoneal exudate contains antivenoms and has bacteriostatic and bactericidal properties (C. M. Volkov, B. K. Shubladze, and others). The peritoneum allocates fibrin, which contributes to the delimitation of the inflammatory process. Therefore even if contaminated with bacteria of the abdominal cavity, peritonitis may not develop or to flow very easily.
There are non-infectious peritonitis, specific infectious and non-infectious (aseptic). The clinical course of Petrograd divided into acute and chronic; by the nature of the effusion in the abdominal cavity - to serous, fibrinous, purulent, putrefactive and hemorrhagic; on the prevalence of inflammatory process on the local and General, distinguished and diffuse.
Local P. often observed at the initial stage of the disease, when the process involves only nearby to the blemish or damaged organ of the peritoneum. Local P. depending on its cause, peculiarities of microbial flora and protective properties of the organism can become widespread, General or limited, encysted, i.e. the abscess of abdominal cavity (pelvic abscesses, subphrenic, interintestinal and so on). Peritonitis caused by the rupture of a hollow organ, called perforating.