Primary and secondary prevention

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And yet adverse external influences affect the changes of the functional state of the nervous system. A violation of the functions of the latter affects the pituitary-adrenal, pituitary-tirednes, of sympathetic-adrenal, immune systems, which play an important role in the adaptation of the organism to the conditions of the environment in which we live.
The system pituitary - adrenal gland produces hormones (oxycorticosteroid)that play an important role in creating a balance between cell membranes and vascular capillaries. These hormones cause the diaphragm seal of every cell and suppress occur in the body, inflammation (for example, joints and spine, myocardium and bronchopulmonary system). In addition, these hormones, as well as the thyroid hormones, are active in the cellular metabolism (metabolism).
In addition, the metabolism role products of sympathetic-adrenal system, such as epinephrine, norepinephrine and their predecessors (dopamine and Dov). These biologically active substances involved in redox processes R cells and are important in maintaining on a certain level of vascular tone. Excessive release of adrenaline, especially in patients with vascular diseases (hypertension, obliterating endarteritis, atherosclerosis of brain vessels, leading to aggravation of these diseases. That is why mental stress, negative emotions are often the most important promoters of sympathetic-adrenal system. When stress increases the secretion of adrenaline, which in this case violates the balance in the body and leads to the aggravation of many diseases.
Infringement of function of one of adaptive systems underlie the development of inflammatory diseases of nature (rheumatic heart disease, arthritis, inflammation of the lungs, kidneys and other organs), Disorders of regulation of functions of several other adaptive systems cause of degenerative diseases (myocardial, arthritis, intervertebral osteochondrosis, atherosclerotic cardiosclerosis). Finally, in violation of the combined functions of adaptive systems of developing the disease, based on functional and degenerative or functional and inflammatory changes in organs and tissues (hypertension, or hypertensive disease, dyskinesia biliary system or intestines, cardiac or angioneurotic form angina, artresources).

the effects of stress on the development of rheumatic heart disease
Fig. 1. The effects of stress on the development of rheumatic heart disease

To realize the essence of various diseases of inflammatory and degenerative nature here is a diagram (Fig. 1), which presents the relationship between stress influences from various unfavorable factors of environment and development of rewma-endocarditis - rheumatic lesions internal (endocardium) and muscle (myocardium) membranes of the heart. As shown in the diagram, under the influence of negative emotions or other stress factors decreases the function of the pituitary-adrenal axis. Mechanism of decreasing function of this system is very complicated. Initially observed, in order to protect the body, pow solution of emission in the internal environment of the body of adrenaline (a substance produced cerebral layer napochechnikov, chromodynamic cells scattered in the gray matter of the brain, the heart), reinforcing adrenocorticotropic (ACTH) the function of the anterior pituitary. The increased content in the internal environment of the body of adrenaline contributes to increased development ACTH, which stimulates the synthesis of glucocorticoids by the adrenal cortex. This process is also serverelement way. Glucocorticoids have the ability to condense the cell membrane, including the myocardium, and thus to keep the intensity of inflammation.
Scientists found that the development of the inflammatory process violated the relationship between the various substrates, which strengthen the connective tissue cells. These violations lead to loosening of the connective tissue, which have a very important qualities in normal functioning of heart muscle. Splitting substrates are mostly carried out under the influence of microbes and their toxins (poisons)that enter the bloodstream during exacerbation of focal infections of the tonsils, carious teeth, sinuses and intestines.