Polverosa - inflammation of the serous membranes multiple cavity (pleural, pericardial, peritoneal, sometimes joints). Polverosa most common tuberculosis and rheumatism, rarely in systemic lupus erythematosus, sepsis, pneumonia, uremia. The clinical course of the disease can be acute (in exudative polyserositis) and chronic (when the adhesive polyserositis).
The clinical picture of polyserositis variable and depends on the number and extent of injury bodies - see Pleurisy, Pericarditis, Peritonitis.

Polverosa (polyserositis) - inflammation of the serous membranes lining the large cavity (pleural, pericardial, peritoneal, sometimes joints). Occurs, in particular, panzeroid (panserositis) - simultaneous inflammation of the serous membranes of all cavities.
Polverosa is a manifestation, or a complication of any other disease. The most common P. tuberculosis and rheumatic origin. Less commonly it occurs in sepsis, pneumonia, typhoid fever, systemic lupus erythematosus, visceral syphilis. Find P. when izotermicheskoi uremia, the scurvy.
Describes polverosa unclear etiology - the so-called periodic disease, family recurring P.
The main role in the occurrence of P. plays giperergicakie reaction serous membranes to infection or toxic effects that causes an increase in vascular permeability and inflammatory changes in serozhnykh cavities.
Polyserositis are exudative and adhesive. In the first case prevail exudative-inflammatory processes. The serous fluid in the cavity can be serous, sero-fibrinous, hemorrhagic or purulent.
When adhesive P. prevail productive-proliferative processes in the form of proliferation of connective tissue, which leads to shrinkage of the serous membranes.
The clinical course of the disease can be acute (in exudative P.) and chronic (with adhesive P.). In most cases adhesive on, are the consequence of exudative process. The clinical picture P. depends on the underlying disease. For acute P. characterized by chest pain, heart or abdomen, fever up to 38-40°, the gradual accumulation of fluid in the cavities.
When the cavities takes a little, it is possible to listen noise of friction pleura, pericardium or even peritoneum. When the accumulation of large quantities of liquid evolving phenomenon of the compression of the relevant authorities (see Pericarditis, Peritonitis, Pleurisy). Duration exudative polyserositis - from several weeks to several months.
After the liquidation of the exudative process remain more or less extensive adhesions or process becomes chronic adhesive polimeraza.
In the clinical picture of periodic disease (see) is dominated by symptoms of acute peritonitis, usually disappear within days. The intervals between attacks from several days to several months.
With very severe phenomena flows described M. N. Acotinum acute infectious diplostraca P. observed in young people in the far East and ended, as a rule, death.
Adhesive polverosa characterized by gradual strengthening of cicatrices, wrinkling, peritoneal, pleural, pericardial. These processes lead to impaired motor function of the intestine to partial or complete obstruction, liver cirrhosis, the deformation of the chest, mediastinal shift, the jugular vein, circulatory failure, the development of big ascites.
Diagnosis of polyserositis is based on the clinical symptoms of pleurisy, pericarditis or peritonitis and typical radiographic changes. For the differential diagnosis should be guided by the symptoms of the underlying disease.
Laboratory tests (blood tests, punctate) help to establish the etiology.
The prognosis depends on the underlying disease, a chronic form - to the degree of development of adhesions and violations functions of the respective bodies.
Treatment is primarily the underlying disease, usually complex.
In non-infectious polyserositis appoint a broad-spectrum antibiotics, taking into account the sensitivity of culture allocated to antibiotics. When P. tubercular etiology conduct combined specific treatment streptomycin (1 g / day), PASK (12 g), ftivazide (1.5 g). When polyserositis rheumatic etiology - Antirheumatic therapy salicylates, antibiotics). When events P. accompanying tuberculosis, rheumatoid arthritis, lupus erythematosus, effective ACTH up to 60 UNITS, cortisone 50-75 mg, prednisolone and prednisone 20 mg per day, reducing allergic and ekssoudativee processes. Shown restorative treatment (vitamins, diet, and others). At considerable accumulation of serous fluid in the cavities - puncture.
Treatment of recurrent disease (aspirin, gistaglobin, nipagin) ineffective.
When adhesive P. conservative treatment is the same, but it gives a less pronounced effect. With significant growths and a violation of the functions of the bodies of surgical treatment (for example, pericardectomy).