Ulcers disease stomach and duodenal ulcer

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Often patients suffering from chronic gastritis, find peptic ulcer disease, which flows hidden. A significant part of the people, on the contrary, initially diagnosed peptic ulcer disease. Clinical manifestation of ulcer disease may be acute or primary chronic, and on its background determine the presence of gastritis. These circumstances, in all probability, depend on the causes of disease, and from various localization of ulcers. Depending on the location of the ulcer and secretory function of the stomach there are three types of stomach ulcers. The plague of the first type is the small curvature, and drink is low, the secretion of hydrochloric acid, while in patients prevails blood group A. the Plague of the second type also localizes the small curvature, combined with duodenal ulcer or accompanied by a narrowing of the pyloric stomach. Gastric secretion in these patients, increased or decreased. Blood group different, at such ulcer marked bleeding, narrowing the janitor, and perforation of the intestine wall, where the plague. The plague of the third type is more often localized in the output part of the stomach, the clinical course is reminiscent of the duodenal ulcer, and is accompanied by increased secretion of hydrochloric acid. These patients prevails zero blood group.
Thus, according to these three fundamental characteristics (localization ulcers, gastric juice secretion, first of all, hydrochloric acid, and blood group) define the characteristics of peptic ulcer disease and complications that are likely to occur. These features of peptic ulcer also indicates that there is a genetic link between the aggressiveness of hydrochloric acid and character of manifestation of dystrophic process in the gastric mucosa, and an important role is played by immunological conflicts that may arise in individuals with different blood groups.
Without diminishing the importance kortiko-visceral component in the development of peptic ulcer disease, you cannot neglect these pathogenetic factors, since they are essential in the course and progression of ulcers, and in the therapeutic effect under the influence of drugs, natural physical and surgical methods of treatment.
In persons with peptic ulcer disease, degenerative changes in the mucous membrane of stomach and duodenal ulcers develop under the influence of various neurohumoral factors, and gastric secretion and acidity increased as mezhpischevaritelny and digestive its phase. In gastric juice increased the content of pepsin and uropathogen. These patients slow evacuation of stomach contents. In addition, with the development of Cicatrical changes in the pylorus of the stomach increases gastric secretion, increases the stagnation of food gruel and are defined using the gastroscope (instrument for research of mucous membrane stomach) morphological signs of gastritis.
It is obvious that in the development of ulcers stomach and duodenal ulcers involving various mechanisms, primarily kortiko-visceral, endocrine and autonomic and local mechanisms of regulation of gastric digestion. Often they are genetically determined and are closely linked. They define clinical forms and variants of ulcers, the propensity to progression and acquired great importance in the successful decision of medical-preventive tasks. Therefore, when deciding issues in a timely diagnosis of peptic ulcer disease and purposeful use of natural factors for secondary prevention is very important to clarify the role of each pathogenetic link and determine whether there are changes not only in the stomach and the duodenum, but also in other organs of digestion.
In patients with gastric ulcer along with gastritis and gastroduodenitis show signs of liver and biliary system, pancreas and intestines. These processes complicate the course of the basic disease. But since they appear mainly through certain time after the beginning of development of ulcer, timely treatment of such patients to prevent the possibility of these opportunistic and associated diseases and especially of various complications - ulcer hemorrhage, perforation of the wall of the stomach or duodenum, and in the presence of adhesive process in the abdominal cavity and the inversion of the intestine.